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Under stress conditions, mitochondrial damage results in the production of excessive mtROS, provoking oxidative damage to mtDNA and mtDNA release into the cytoplasm through the mitochondrial permeability transition pore, which further activates the DNA sensor cGAS-STING and facilitates downstream inflammatory processes (101, 102)

Franco R., Li S., RodriguezRocha H., et al., Molecular Mechanisms of Pesticideinduced Neurotoxicity: Relevance to Parkinson's Disease, ChemicoBiological Interactions 188, no