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Description

& Turkheimer, F

Of these, three genes were newly discovered, thus, further follow-up studies are warranted for their functional annotation

Rumack BH, Bateman DN

This multifaceted disruption of redox homeostasis significantly enhances DLBCL cell vulnerability to ferroptosis and provides a mechanistic rationale for exploiting BTK inhibitors in combination with ferroptosis-inducing therapies in high-risk, apoptosis-refractory lymphomas