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62 Macrophages exert atheroprotective effects under homeostatic conditions, through both the clearance of lipoprotein by endocytosis and apoptotic cells by phagocytosis or efferocytosis, thereby abolishing the inflammatory processes involved in plaque formation

In previous studies using an NQO2 knockdown cell line and NQO2 siRNA, the inhibition of NQO2 activity induced the upregulation of antioxidant enzymes, which resulted in increased cellular resistance to oxidants and protected cellular components from oxidation-related damage 56,57,58

( C ) The decreased GSH:GSSG increases LDH release, consistent with activation of oxidatively driven cell death ( n = 6)