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So far, there has been only one potential drug candidate for preventing the development of oxaliplatin-induced acute and delayed CIPN, namely, calmangafodipir, a mitochondrial manganese superoxide dismutase mimetic, which is currently being studied in a placebo-controlled, double-blinded randomized phase III study [24]

Cystathionine Beta Synthase-Hydrogen Sulfide System in Paraventricular Nucleus Reduced High Fatty Diet Induced Obesity and Insulin Resistance by Brain-Adipose Axis

This type of invasion can compromise essential functions such as swallowing, speech, and breathing, making it crucial to balance tumor removal with functional preservation and quality of life

mtDNA proximity to these ROS production sites makes them more vulnerable to damage, mutations, deletions, and genetic instability [16]