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These findings, together with various oligomerization states of VP35 in different filoviruses 16 , imply that the differences in VP35 dynamics among the filoviruses may dictate the potential divergent mechanisms of replication and pathogenesis during viral propagation

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Purinergic Signalling 2011

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2004 Mar;38(3):218-24

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In contrast, iNOS, expressed in response to inflammation, produces micromolar quantities of NO (Nathan and Xie, 1994)

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