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Macrophages internalize pathogens through phagocytosis, then kill them and digest them in phagosomes ( + levels in a CD38-dependent manner, resist pathogen-induced changes in macrophage morphology and the F-actin cytoskeleton distribution, and reduce the ability of non-conditioned Salmonella to infect macrophages ( + supplementation reversed the morphological transformation of macrophages and the accumulation of dorsal F-actin, and restored the ability of Salmonella to infect macrophages in the presence of LXR agonists

The study found that NNMT inhibitors "reduced intracellular 1-MNA, increased intracellular NAD+ and S-(5-adenosyl)-L-methionine (SAM), and suppressed lipogenesis." This metabolic improvement occurs within days of starting treatment, before any weight loss becomes measurable