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As a consequence, an in-depth exploration of how exercise affects its therapeutic potential at these physiological levels, in particular concerning the mechanisms of oxidative stress and the HPA axis, is vital for a deeper insight of the potential of exercise in PTSD treatment

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Because roGFP2-Orp1 undergoes a specific H 2 O 2 -dependent oxidation, but can also be reduced, its redox state is influenced either by the level of the oxidant (H 2 O 2 ) or by the reductants themselves, such as GSH and thioredoxin [15]

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