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FIGURE 3 6.1 Anti-hepatic fibrosis effect of curcumin PPAR (NR1C1), a ligand-responsive nuclear hormone receptor, demonstrates pronounced hepatic tissue enrichment, was originally characterized as a pharmacological target for heterogenin that induces peroxisome proliferation in rodents (Issemann and Green, 1990)

muciniphila , resulting in a significant decrease of its relative abundance in APAP_BSL group, but this phenomenon did not mean that A

Excessive extracellular glutamate concentration also impacts systemic xc - , preventing cystine entry, which in turn leads to GSH depletion triggering ferroptosis [95]

This process involves the breakdown of carbohydrates, such as glucose, into smaller molecules to generate ATP, which serves as a primary energy source for various cellular functions