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Critically, SCFAs are master epigenetic regulators, acting as inhibitors of the enzyme histone deacetylase (HDAC), which is involved in the histone modification [194, 195]

Collectively, these findings indicate that FA can counteract erastin-induced ferroptosis and ameliorate neuroinflammation primarily through modulation of the Nrf2/GPX4 pathway, highlighting its potential as a candidate therapeutic for Alzheimers disease

Importantly, although several chemically-distinct agents, including glutamate, erastin, sulfasalazine and BSO, can all induce ferroptosis through GSH depletion, the present study extends our previous work by demonstrating that these diverse upstream perturbations converge on a shared downstream signaling axis centered on PDI

In 3xTg-AD mice, human dental pulp stem cell transplantation exerts antioxidative stress and neuroprotective effects (Xiong et al., 2024)