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As MOTS-c activates AMPK and NRF2, it also indirectly suppresses the mTOR (mechanistic target of rapamycin) pathway, the master growth and anabolism regulator that, when chronically overactivated, accelerates the accumulation of senescent cells

doi:10.1016/S0021-9258(19)44924-7

Notably, pathways in the combination group presented a greater degree of overlap with those in the control group than those in the single-natural product groups did, indicating that this combination regulated more AD-related pathways

Semax, for example, emerged from the observation that ACTH(4-10) retains neurotrophic activity without the corticotropic activity of ACTH(1-39)