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The second antigen, small nuclear ribonuclear polypeptide A, also known as U-1A, is a 70-kD peptide recognized by anti-ribonuclear protein autoantibodies associated with mixed connective tissue diseases (i.e., arthralgias, Raynauds phenomenon, arthritis, myositis, pulmonary fibrosis, and sclerodactyly [71, 72]

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This nephropathy originates from diabetes-induced dysregulation of glucose metabolism, which in turn triggers a series of metabolic abnormalities, hemodynamic changes, inflammatory responses, and fibrotic processes that ultimately drive disease progression (Tuttle et al., 2022)

Evidence for mitochondrial uptake of glutathione by dicarboxylate and 2-oxoglutarate carriers